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Dihydrotestosterone: An Introduction
There are a lot of different hormones in your body, and these hormones to a number of different things in order to help your body function properly. Testosterone help provide your body with sexual hormones, human growth hormone helps give your body energy and keeps your cells strong, even estrogen has some effect in your body so when you find out that you are suffering from Dihydrotestosterone related hair loss, you are likely wondering what Dihydrotestosterone is.
Like testosterone, Dihydrotestosterone is an androgen, also known as a male hormone. Dihydrotestosterone is actually created from testosterone combining with a common enzyme in your body. If you are a male, you likely experienced great bursts of Dihydrotestosterone when you are in your mother's womb. That is because Dihydrotestosterone is one of the leading hormones for male physical growth in the embryonic and fetal stages of development.
Once you get older, however, Dihydrotestosterone actually has no real purpose in your body anymore. It has a few very small functions, but nothing that important or relevant. In general, it builds up in your body very slowly and moves around until it eventually disappears. However, it is also possible that your body may start creating Dihydrotestosterone at a faster pace. When this occurs, the Dihydrotestosterone has absolutely no where to go and nothing to do. So would it ends up doing is binding to your hair follicles, preventing them from receiving very important nutrients that they need in order to stay alive.
This is what causes the hair loss - nutrients are unable to reach your hair follicles, and the hairs will slowly begin to weaken and die. The longer you wait to treat your Dihydrotestosterone related hair loss, the more likely it is that this hair loss becomes a permanent problem. In addition to causing hair loss, Dihydrotestosterone has also been linked to other health problems like prostate cancer.
"Dihydrotestosterone." Dihydrotestosterone. Dihydrotestosterone, Web. 09/15/2011.